Just eat meat.

Created by Michael Goldstein (@bitstein)



Dr. Thomas Seyfried

Travis Christofferson, MS

Dr Richard David Feinman & Dr Eugene Fine

Miriam Kalamian

Dr. Nasha Winters ND FABNO L.Ac Dipl.OM

Dr. Colin Champ

Dr. Dawn Lemanne

Dominic D’Agostino, Ph.D.

Dr. Sebastian Winter

Angela Poff PhD – University of South Florida

Andrew Koutnik, M.S.

Dr. Adrienne Scheck

Dr. John Bergman

Dr. Darren Schmidt

Paleomedicina - Dr. Csaba Tóth, Dr. Zsófia Clemens

Nathan Ward, PhD at Moffett Cancer Center

Tucker Goodrich


PI 3-Kinase


Benefits of Ketogenic Diet

Anecdotes / Case Studies


Studies In Progress

Squamous Cell Carcinoma / Head and Neck Cancer

From the first article linked here about TMAO, initial questions immediately rise:

  1. Correlation should never be confused with causation. If A is correlated with B, three conclusive possibilities exist:
    1. A caused B
    2. B caused A
    3. C variable(s) could be interacting with both A and B causing the correlation.
  2. TMAO is naturally high in fish which is largely associated with heart disease protection. If TMAO is a cause of heart disease, high fish consumption should be associated with heart disease. As of time of this writing, no association exists.
  3. The gut bacteria responsible for converting more TMAO appear to be the real culprit. Could TMAO levels be the smoke and not the fire? What other factors are involved? Does chronic or excessive red meat consumption lead to higher levels of TMAO-converting bacteria? Does a low-fiber (low-vegetable) diet play a role? Intake of whole grains? Chronic infection? Other causes of gut dysbiosis? For instance, Paul Jaminet, PhD remarks in “Lessons From The Latest Red Meat Scare”:

    “…the gut flora is a much better predictor of blood TMAO levels than whether someone eats meat. Those with high Prevotella, low Bacteroides averaged about triple the TMAO levels of those with low Prevotella, high Bacteroides flora.”


Created by Travis Statham - @travis_statham January 14, 2019